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Comparing the BSI group with the non-BSI group, in the first group we notice a greater incidence of HLC pair suppression (both severe and extreme) (p=0

Comparing the BSI group with the non-BSI group, in the first group we notice a greater incidence of HLC pair

Continue readingComparing the BSI group with the non-BSI group, in the first group we notice a greater incidence of HLC pair suppression (both severe and extreme) (p=0

While allowing era of book sequences that focus on self-antigens, in addition, it prevents the era of man made sequences that could contain high degrees of T-cell epitopes and become strongly immunogenic

While allowing era of book sequences that focus on self-antigens, in addition, it prevents the era of man made sequences

Continue readingWhile allowing era of book sequences that focus on self-antigens, in addition, it prevents the era of man made sequences that could contain high degrees of T-cell epitopes and become strongly immunogenic

In addition to the most abundant amyloid protein, LMD-LC-MS also identified serum amyloid P, apolipoprotein E, and apolipoprotein A-IV, which are associated with the amyloid formation in the amyloid deposits (Table 3, labeled underline) [23]

In addition to the most abundant amyloid protein, LMD-LC-MS also identified serum amyloid P, apolipoprotein E, and apolipoprotein A-IV, which

Continue readingIn addition to the most abundant amyloid protein, LMD-LC-MS also identified serum amyloid P, apolipoprotein E, and apolipoprotein A-IV, which are associated with the amyloid formation in the amyloid deposits (Table 3, labeled underline) [23]

Taken together, these results show that immunoglobulins, including IgA, found in renal tissues may be secondary phenomena derived from systemic immune reactions rather than primary etiologic agents [34]

Taken together, these results show that immunoglobulins, including IgA, found in renal tissues may be secondary phenomena derived from systemic

Continue readingTaken together, these results show that immunoglobulins, including IgA, found in renal tissues may be secondary phenomena derived from systemic immune reactions rather than primary etiologic agents [34]